Redefining electrosensitivity: A new literature-supported model

Author: Mary Redmayne1,2, Siobhan Reddel3
1 SGEES, Victoria University of Wellington , New Zealand.
2 Monash University , Melbourne, Australia.
3 Budja Budja Aboriginal Co-operative.
Conference/Journal: Electromagn Biol Med
Date published: 2021 Jan 25
Other: Special Notes: doi: 10.1080/15368378.2021.1874971. , Word Count: 246

In critically examining literature on electrohypersensitivity and the reported somatic responses to anthropogenic modulated radiofrequency radiation (RFR) exposure, it becomes apparent that electrohypersensitivity is one part of a range of consequences. Current evidence on the necessity of considering patients' overall health status leads us to propose a new model in which electrohypersensitivity is but part of the electrosensitive status inherent in being human. We propose the likelihood and type of response to environmental RFR include i) a linear somatic awareness continuum, ii) a non-linear somatic response continuum, and iii) the extent of each individual's capacity to repair damage (linked to homeostatic response). We anticipate this last, dynamic, aspect is inextricably linked to the others through the autonomic nervous system. The whole is dependent upon the status of the interconnected immune and inflammatory systems. This holistic approach leads us to propose various outcomes. For most, their body maintains homeostasis by routine repair. However, some develop electrohypersensitivity either due to RFR exposure or as an ANS-mediated, unconscious response (aka nocebo effect), or both. We suggest RFR exposure may be one factor in the others developing an auto-immune disease or allergy. A few develop delayed catastrophic disease such as glioma. This model gives the blanket term ElectroMagnetic Illness (EMI) to all RFR-related conditions. Thus, EHS appears to be one part of a range of responses to a novel and rapidly changing evolutionary situation.

Keywords: Autonomic Nervous System; Electrohypersensitivity; NIR; electrosensitivity; homeostasis; immune dysregulation; inflammation; nocebo; non-ionising radiation.

PMID: 33492997 DOI: 10.1080/15368378.2021.1874971